e t aristizabal prada ludwig-maximilians university of munich | The role of GSK3 and its reversal with GSK3 antagonism in e t aristizabal prada ludwig-maximilians university of munich Tropomyosin receptor kinase (Trk) inhibitors are investigated as a novel targeted therapy in various cancers. We investigated the in vitro effects of the pan-Trk inhibitor GNF-5837 in .
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0 · Tropomyosin receptor kinase: a novel target in screened
1 · Tropomyosin receptor kinase: a novel target in screened
2 · The role of GSK3 and its reversal with GSK3 antagonism in
3 · Targeted therapy of gastroenteropancreatic neuroendocrine
4 · Preclinical in vitro investigation to evaluate novel molecular
5 · Elke Tatjana Aristizabal Prada
6 · Comparative Genomics and Transcriptome Profiling in Primary
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Molecular targeted therapy of advanced neuroendocrine tumours (NETs) of the gastroenteropancreatic (GEP) system currently encompasses approved therapy with the .E T Aristizabal Prada and C J Auernhammer. Department of Internal Medicine IV, Campus Grosshadern, University-Hospital, Ludwig-Maximilians-University of Munich, Munich, . Abstract. Molecular targeted therapy of advanced neuroendocrine tumours (NETs) of the gastroenteropancreatic (GEP) system currently encompasses approved therapy with the . In the first part of this study, effects of the highly selective pan-Trk inhibitor GNF-5837 (Albaugh et al. 2012) were investigated in human neuroendocrine tumor cell lines in vitro. .
Aristizabal Prada ET, Reuther C, Young K, Korbonits M, Göke B, Grossman A, Auernhammer CJ.Tropomyosin receptor kinase (Trk) inhibitors are investigated as a novel targeted therapy in various cancers. We investigated the in vitro effects of the pan-Trk inhibitor GNF-5837 in .
It has previously been reported that inhibition of GSK3 restored autophagy (Weikel et al. 2016, Zhang et al. 2016, Aristizabal Prada et al. 2017) and re-sensitized different tumor . The genetic basis of the four familial forms of primary aldosteronism (familial hyperaldosteronism FH types I-IV) and the majority of sporadic unilateral aldosterone .
Tropomyosin receptor kinase: a novel target in screened
Elke Tatjana Aristizabal Prada is an academic researcher from Ludwig Maximilian University of Munich. The author has contributed to research in topic(s): Everolimus & Viability assay. The .E T Aristizabal Prada, 548 V Heinzle et al Tropomyosin receptor kinase (Trk) in NETs Endocrine-Related 25:5 Cancer Introduction Neuroendocrine tumors (NETs) are highly heterogeneous .Molecular targeted therapy of advanced neuroendocrine tumours (NETs) of the gastroenteropancreatic (GEP) system currently encompasses approved therapy with the mammalian target of rapamycin (mTOR) inhibitor everolimus .
E T Aristizabal Prada and C J Auernhammer. Department of Internal Medicine IV, Campus Grosshadern, University-Hospital, Ludwig-Maximilians-University of Munich, Munich, Germany. Correspondence should be addressed to C J Auernhammer: [email protected]. Abstract.
Abstract. Molecular targeted therapy of advanced neuroendocrine tumours (NETs) of the gastroenteropancreatic (GEP) system currently encompasses approved therapy with the mammalian target of rapamycin (mTOR) inhibitor everolimus . In the first part of this study, effects of the highly selective pan-Trk inhibitor GNF-5837 (Albaugh et al. 2012) were investigated in human neuroendocrine tumor cell lines in vitro. GNF-5837 significantly decreased GOT1 cell survival (Fig. 1 .Aristizabal Prada ET, Reuther C, Young K, Korbonits M, Göke B, Grossman A, Auernhammer CJ.Tropomyosin receptor kinase (Trk) inhibitors are investigated as a novel targeted therapy in various cancers. We investigated the in vitro effects of the pan-Trk inhibitor GNF-5837 in human neuroendocrine tumor (NET) cells.
It has previously been reported that inhibition of GSK3 restored autophagy (Weikel et al. 2016, Zhang et al. 2016, Aristizabal Prada et al. 2017) and re-sensitized different tumor entities to chemotherapy, radiotherapy or targeted therapy (Domoto et al. 2016). The genetic basis of the four familial forms of primary aldosteronism (familial hyperaldosteronism FH types I-IV) and the majority of sporadic unilateral aldosterone-producing adenomas has now been resolved. Familial forms of hyperaldosteronism are, however, rare.Elke Tatjana Aristizabal Prada is an academic researcher from Ludwig Maximilian University of Munich. The author has contributed to research in topic(s): Everolimus & Viability assay. The author has an hindex of 10, co-authored 13 publication(s) receiving 171 citation(s).
E T Aristizabal Prada, 548 V Heinzle et al Tropomyosin receptor kinase (Trk) in NETs Endocrine-Related 25:5 Cancer Introduction Neuroendocrine tumors (NETs) are highly heterogeneous tumors originating from distinct cell precursors ( Schimmack et al. 2011). Current data show an increase in incidence of gastroenteropancreatic neuroendocrine tumorsMolecular targeted therapy of advanced neuroendocrine tumours (NETs) of the gastroenteropancreatic (GEP) system currently encompasses approved therapy with the mammalian target of rapamycin (mTOR) inhibitor everolimus .E T Aristizabal Prada and C J Auernhammer. Department of Internal Medicine IV, Campus Grosshadern, University-Hospital, Ludwig-Maximilians-University of Munich, Munich, Germany. Correspondence should be addressed to C J Auernhammer: [email protected]. Abstract.
Abstract. Molecular targeted therapy of advanced neuroendocrine tumours (NETs) of the gastroenteropancreatic (GEP) system currently encompasses approved therapy with the mammalian target of rapamycin (mTOR) inhibitor everolimus .
In the first part of this study, effects of the highly selective pan-Trk inhibitor GNF-5837 (Albaugh et al. 2012) were investigated in human neuroendocrine tumor cell lines in vitro. GNF-5837 significantly decreased GOT1 cell survival (Fig. 1 .
Aristizabal Prada ET, Reuther C, Young K, Korbonits M, Göke B, Grossman A, Auernhammer CJ.Tropomyosin receptor kinase (Trk) inhibitors are investigated as a novel targeted therapy in various cancers. We investigated the in vitro effects of the pan-Trk inhibitor GNF-5837 in human neuroendocrine tumor (NET) cells. It has previously been reported that inhibition of GSK3 restored autophagy (Weikel et al. 2016, Zhang et al. 2016, Aristizabal Prada et al. 2017) and re-sensitized different tumor entities to chemotherapy, radiotherapy or targeted therapy (Domoto et al. 2016).
The genetic basis of the four familial forms of primary aldosteronism (familial hyperaldosteronism FH types I-IV) and the majority of sporadic unilateral aldosterone-producing adenomas has now been resolved. Familial forms of hyperaldosteronism are, however, rare.Elke Tatjana Aristizabal Prada is an academic researcher from Ludwig Maximilian University of Munich. The author has contributed to research in topic(s): Everolimus & Viability assay. The author has an hindex of 10, co-authored 13 publication(s) receiving 171 citation(s).
Tropomyosin receptor kinase: a novel target in screened
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The role of GSK3 and its reversal with GSK3 antagonism in
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e t aristizabal prada ludwig-maximilians university of munich|The role of GSK3 and its reversal with GSK3 antagonism in
